The damage to the granular cells and the expression of CIRBP, RBM3, HSP70, HIF-1, AIF1, SIRT1, Ngb, cFos, p53, and CCC9 in postmortem human cerebellas from people who died by hanging, strangling, drowning, suffocation by anaphylaxis, or aspirating food may be closely linked to the length of the agony.
The findings of the current study may suggest that brains taken from acutely deceased people retain their shape and are in a signal pathway scenario to defend against hypoxic brain damages rather than in a phase from apoptotic to cell death.
The brain tissues without blood reperfusion may be used and trusted to assess and investigate the microscopic functioning of the cells. brain damage due to hypoxia antigens linked to hypoxia, Immunohistochemistry, acute hypoxia alterations.
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